Prostate cancer is AR-dependent; castration-resistant progression often involves AR amplification/splice variants, DDR defects, and lineage plasticity. We provide AR/PSA/PSMA biomarker antibodies plus AR antagonists, PARP, and AKT probes for resistance, metabolism, and combinations.
Focus areas: AR signaling & splice variants, PARP in DDR defects, neuroendocrine transdifferentiation, bone metastasis niche
Commonly used for mechanistic studies; follow your lab SOP, compound datasheets, and ethics approvals.
| Compound | Targets / pathways | Experimental notes |
|---|---|---|
| Enzalutamide | AR (antagonist) | CRPC probe; seizure threshold/fatigue monitoring. |
| Abiraterone | CYP17 (androgen synthesis) | Adrenal androgen suppression; electrolytes/LFTs. |
| Apalutamide | AR (antagonist) | nmCRPC mapping; rash/thyroid function. |
| Olaparib | PARP1/2 | Synthetic lethality in HRR defects; marrow suppression. |
| Talazoparib | PARP1/2 (potent) | Potent DDR probe; higher hematologic toxicity. |
| Ipatasertib | AKT1/2/3 | PTEN-loss PI3K/AKT validation; diarrhea/hyperglycemia. |
| Darolutamide | AR antagonist (structurally distinct) | Lower CNS penetration—useful comparator. |